This observation aligns with the viral protein synthesis dynamics in which PB2, PB1, PA, and NP segments are expressed early while HA, NA, M1, and M2 are produced relatively late in infection (6, 10, 43), highlighting that IAV utilizes hnRNPM to differentially regulate late gene transcription, thereby promoting efficient virus multiplication in human cells. The gene discussed is XK; the disease is infection.