Myeloid cell trafficking to tumours depends on PI3Kγ‐mediated integrin activation, and subsequent myeloid cell recruitment and tumour inflammation depend on PLCγ, CalDAG‐GEFI and II, Rap1a, RIAM, talin, paxillin and myosin light chain kinase [65, 66]. The gene discussed is APBB1IP; the disease is neoplasm.