For example, TTBK2 phosphorylates M‐phase phosphoprotein 9 (MPP9), promoting its ubiquitination and degradation, which facilitates the removal of CP110 and CEP97 from the mother centriole, enabling ciliary initiation.[31] Similarly, ASK1‐mediated phosphorylation stabilizes HDAC6 by preventing its ubiquitination, promoting ciliary disassembly in retinopathy of prematurity.[11]. This evidence concerns the gene MPHOSPH9 and retinopathy of prematurity.