Tumor‐associated neutrophils (TANs) can be phenotypically classified into proinflammatory N1 and anti‐inflammatory N2 subpopulations, which play distinct roles in tumor progression.[26] Cell scoring further showed that the N1‐like neutrophil phenotype was highly activated in APM‐treated cells with downregulation TGF‐β signaling, while APM‐naïve cells exhibited the N2‐like neutrophil phenotype (Figure 5E). Here, TGFB1 is linked to neoplasm.