Notably, AXL is highly expressed in undifferentiated, medullary, and papillary thyroid carcinomas.[27] PROS1 expression is significantly correlated with lymph node staging in patients with PTC, and downregulation of PROS1 inhibits PTC cell proliferation and migration.[28] While these findings underscore the relevance of TAM receptor signaling in PTC, many questions remain unanswered, particularly regarding the full extent of TAM receptor signaling in the PTMC tumor microenvironment. Here, AXL is linked to neoplasm.