Specifically, LLPK intervenes in biological processes such as lipogenesis, fatty acid oxidation, and fatty acid transport in mice livers by regulating the expression of key downstream factors of PPARα (Scd1, Acox1, Acaa1b, Slc27a1, Acsl1, and Ehhadh), thus improving lipid metabolic disorders (Figure 7). The gene discussed is ACSL1; the disease is Disorder of lipid metabolism.