In contrast to parvalbumin-positive and calretinin-positive interneurons, which in prior studies have shown contradictory results to either remain stable, increase, or decrease in epilepsy six weeks after kainic acid-induced status epilepticus, we found a significant increase in NECAB1-positive cell density in the amygdala bilaterally and the paraventricular nucleus of thalamus of the epileptic control group compared to sham-operated animals. This evidence concerns the gene NECAB1 and epilepsy.