While some studies support its function as a key modulator of peripheral eosinophil homeostasis and tissue eosinophilia [39,40], contrasting evidence from CCL11-deficient murine models suggests that CCL11 is dispensable for airway eosinophil recruitment, potentially due to compensatory mechanisms involving alternative chemokine pathways [41]. Here, CCL11 is linked to Increased total eosinophil count.