BMAL1 and myocardial infarction: The molecular mechanism is closely related to Histone Deacetylase 3 (HDAC3), a crucial element of the circadian negative feedback loop, which upregulates BMAL1 to restore mitochondrial autophagy by regulating the REV-ERBα/BMAL1 pathway, thereby alleviating myocardial infarction injury [82].