It is noteworthy that certain biological pathways identified as enriched in this study, including taste transduction and nicotine addiction, might not represent a direct relevance to the biology of SCLC or to the genes TSPAN8, UCHL1, and MYC, which were identified as potential targets of RG6016 through our bioinformatic and molecular docking analyses. The gene discussed is MYC; the disease is nicotine dependence.