Prostate tumors and normal PSMA-expressing tissues (the parotid glands, liver, spleen, and kidneys) showed similar biodistribution patterns for 18F-PSMA-1007 and 68Ga-PSMA-11 [101], but 18F-PSMA-1007 demonstrated a statistically significant increase in SUV uptake in intraprostatic lesions relative to the uptake by the urinary bladder. Here, FOLH1 is linked to prostate neoplasm.