Another study corroborated the enhancement of ERK signalling by UCA1 and revealed that CUDR, an alternative transcript variant of UCA1, could promote the migration and invasion of pancreatic cancer cells by activating AKT/FAK and ERK signalling to induce an epithelial-to-mesenchymal transition (EMT) [96,97]. This evidence concerns the gene UCA1 and pancreatic neoplasm.