The dysfunction of this gene loci could lead to gene expression changes in anti-NMDAR encephalitis, such as the increased survival of autoreactive B-cells and increased anti-NMDAR antibody production (GluN1 subunit) and pro-inflammatory cytokine production, while the chromosome 11 loci findings have an impact on the inflammatory gene expression balance, immune regulation, and antigen processing. This evidence concerns the gene GRIN1 and viral encephalitis.