GRIN1 and encephalitis: The dysfunction of this gene loci could lead to gene expression changes in anti-NMDAR encephalitis, such as the increased survival of autoreactive B-cells and increased anti-NMDAR antibody production (GluN1 subunit) and pro-inflammatory cytokine production, while the chromosome 11 loci findings have an impact on the inflammatory gene expression balance, immune regulation, and antigen processing.