In contrast, NKR‐P1A+ and NKR‐P1A‒ subsets in total NK cells and adaptive NKG2C+ NK cells in CMV seropositive individuals exhibited vastly different transcriptomes, highlighting the differential impact of CMV infection on NK cells expressing NKR‐P1A receptor (Figure 6E,F). The gene discussed is KLRC2; the disease is cytomegalovirus infection.