In contrast, NKR‐P1A+ and NKR‐P1A‒ subsets in total NK cells and adaptive NKG2C+ NK cells in CMV seropositive individuals exhibited vastly different transcriptomes, highlighting the differential impact of CMV infection on NK cells expressing NKR‐P1A receptor (Figure 6E,F). Here, KLRB1 is linked to cytomegalovirus infection.