It has been shown that AT2 cell injury in a mouse model resulted in ‘C-C Motif Chemokine Receptor 2’ (CCR2)-mediated accumulation of non-resident CD11b+ macrophages and ‘lymphocyte antigen 6 family member C’ (Ly6C)hi monocytes, driving lung fibrosis.120. Here, CCR2 is linked to pulmonary fibrosis.