CFL1 and infection: This enhanced fluid-phase endocytosis in response to LD-R-infection was further confirmed by structural illumination microscopy (SIM) imaging, which showed increased Cofilin turnover with dispersed distribution in LD-R-infected-KCs, leading to enhanced F-Actin rearrangement and protrusion formation (Popow-Woźniak et al., 2012), which was absent for uninfected or LD-S-infected-KCs (Figure 4Ci, ii, Di, ii).