Apart from these two amyloidogenic peptides, it has recently been shown that a familial mutant of the alpha-synuclein protein (A53T), the misfolding and aggregation of which is linked to Parkinson’s disease, seeds are intracellularly preferentially on mitochondrial membrane surfaces, with CL being sequestered within the aggregating lipid–protein complexes [67]. The gene discussed is SNCA; the disease is Parkinson disease.