IL6 and osteochondritis dissecans: Garamszegi et al. [15] found that elevated IL-6 was observed in the OB of patients with AD, and that the presence of IL-6 and cyanobacterial toxin (β-Methylamino-L-alanine [BMAA]) in the olfactory microenvironment may exacerbate olfactory neuronal damage, leading to OD.