This is interesting because EBV infection has been reported to uniquely trigger high levels of IL‐18, IFNγ‐induced chemokines, and vastly expanded CD38+HLA–DR+ T lymphocytes,46 a subset of (pathogenic) T cells that has been associated with IFNγ production in MAS.38 The gene discussed is IL18; the disease is Epstein-Barr virus infection.