For example, lncRNA PRNCR1 enhanced the proliferation of breast cancer cells and suppressed apoptosis.7 In addition, lncRNA MALAT1 inhibited cisplatin sensitivity of NSCLC through regulating the microRNA/miRNA (miR)-197-3p/Catenin axis.8 Notably, abnormally expressed lncRNAs are closely associated with the pathogenesis of cancers, including NSCLC. Here, MALAT1 is linked to non-small cell lung carcinoma.