Dual-luciferase reporter, chromatin immunoprecipitation (ChIP), and Western blotting studies uncovered that steady overexpression or silencing of FOXC1 into NB cells led to an increase or decrease in FOXC1 activity (Fig. S1E), FOXC1 enrichment (Fig. 1G), and protein levels of ASNS or GPT2 (Fig. 1H and Fig. S1F). Here, GPT2 is linked to neuroblastoma.