In EDTA-PTCP, the platelet cell membrane has undergone structural changes, resulting in antibodies binding to genetically mutated antigens known as “neoantigens.” Glycoproteins (GP) IIb or I have been suggested to be the neoantigen in PTCP, as platelets of individuals with Glanzmann thrombasthenia do not interact with these antibodies [7]. The gene discussed is ART4; the disease is Glanzmann thrombasthenia 1.