In EDTA-PTCP, the platelet cell membrane has undergone structural changes, resulting in antibodies binding to genetically mutated antigens known as “neoantigens.” Glycoproteins (GP) IIb or I have been suggested to be the neoantigen in PTCP, as platelets of individuals with Glanzmann thrombasthenia do not interact with these antibodies [7]. This evidence concerns the gene ITGA2B and Glanzmann thrombasthenia 1.