The HDAC inhibitor Panobinostat was used as a positive control for apoptosis because of its potent ability to induce apoptosis in lymphoma cell lines at high concentrations [51].These data therefore reveal a metabolic vulnerability of EBV-infected LCLs, where simultaneous SCD1 and FADS2 inhibition hypersensitizes LCLs to apoptosis from physiologically-relevant concentrations of palmitate [50]. This evidence concerns the gene FADS2 and lymphoma.