SHANK3 and neuroleptic malignant syndrome: TAF1 and DPM2 provide potential clues about parkinsonism and increased creatine phosphokinase in neuroleptic malignant syndrome, while abnormalities in RALGPS1 suggest links to both anti-NMDAR antibody encephalitis and the SHANK3 gene, which is known to predispose to catatonia.