The present study adds to our previous characterizationof heterobivalent(SST2/albumin) radioligands, named NODAGA-cLAB-TATEs, with the primary aim of identifying the mechanistic basis for theincreased integral tumor uptake of [64Cu]­Cu-NODAGA-cLAB4-TATE compared to [64Cu]­Cu-NODAGA-TATE. The gene discussed is ALB; the disease is neoplasm.