MLXIPL and steatosis: H19 promotes steatosis and lipid accumulation by modulating several pathways, including the miR-130a/peroxisome proliferator-activated receptor gamma (PPARγ) axis, MLX-interacting protein-like (MLXIPL) expression, and mechanistic target of rapamycin complex 1 (mTORC1) signaling [35, 36].