TNFRSF12A and urinary bladder cancer: Biological pathways involved in TNFRSF12A were identified by GSEA, and the samples in TCGA-STAD cohort were split into high and low groups according to the median expression level of TNFRSF12A. KEGG enrichment analysis demonstrated that the high expression group mainly participated in the pathways of Bladder cancer, extracellular matrix (ECM)-receptor interaction, Hepatitis C, Proteasome, and Virion-Hepatitis viruses (Figure 3A).