COL1A1 and fatty liver disease: Subsequent histological analyses, including H&E staining, Sirius Red staining, Oil Red O staining (Figure 3A), as well as IHC for fibrosis markers (α‐SMA, COL1A1) and infiltrating macrophage markers F4/80 (Figure 3B and Figure S4B, Supporting Information), demonstrated significant improvements in hepatic steatosis, inflammation, and fibrosis in the GSN‐supplemented group compared to the AAV8 empty vector group.