In the context of chicken coccidia infections, Cornelissen et al. reported that, at 4 days post-infection (dpi) with E. maxima, acervulina and tenella, there was a suppression of IFN-γ mRNA across different intestinal segments (jejunum/duodenum/cecum), along with increased expression of IL-4/IL-10 [12]. Here, IL10 is linked to infection.