MTOR and breast cancer: Recent studies have revealed a novel function of CSNK1G2 in regulating oxidative stress response and modulating reactive oxygen species levels.[38] Additionally, CSNK1G2 has been shown to play a pivotal role in sensitizing breast cancer cells to tamoxifen (TAM) toxicity by differentially regulating the PI3K/AKT/mTOR/S6K and ERK signaling pathways.[39] Despite these advancements, the association between CSNK1G2 and neonatal respiratory distress remains unexplored.