This cascade ultimately enhances the transcription of genes associated with cell cycle progression, such as cyclin D1, c‐Myc, c‐Fos, c‐Jun, Elk‐1, and cAMP‐response element binding protein (CREB), thereby contributing to increased tumor cell proliferation [50, 57, 58, 59, 60, 61]. This evidence concerns the gene MYC and neoplasm.