In the Eg.P29/Myc-BioID2-MCS group, the enriched biological functions were cadherin binding, actin binding, ATPase activity, actin filament-binding, and tubulin binding (Figure 1E), and the enriched KEGG pathways were amyotrophic lateral sclerosis, RNA transport, endocytosis, Salmonella infection, and pathogenic E.coli infection (Figure 1F). This evidence concerns the gene MYC and Salmonella Infections.