MYC and amyotrophic lateral sclerosis: In the Eg.P29/Myc-BioID2-MCS group, the enriched biological functions were cadherin binding, actin binding, ATPase activity, actin filament-binding, and tubulin binding (Figure 1E), and the enriched KEGG pathways were amyotrophic lateral sclerosis, RNA transport, endocytosis, Salmonella infection, and pathogenic E.coli infection (Figure 1F).