The study by Meng et al. also revealed the crucial role of the fibroblast growth factor (FGF) signaling pathway in the recurrence of RA, with a notable increase in expression levels in the lining FLSs of recurrent RA patients.93 Further research indicated that knocking out FGF10 or inhibiting FGF receptor 1 (FGFR1) effectively regulated the activity of the FGF pathway, reducing bone erosion, and offering new hope for the remission of recurrent RA.93 The gene discussed is FGFR1; the disease is rheumatoid arthritis.