LGALS8 and acute kidney injury: Through these interactions, Gal-8 may modulate diverse cellular processes that influence adaptive or maladaptive tissue repair following AKI, including epithelial cell proliferation, survival and EMT (Oyanadel et al. 2018; Perez-Moreno et al. 2024b), as well as immune responses that shape inflammation (Pardo et al. 2017; Obino et al. 2018; Prato et al. 2020; Zick 2022; Prato et al. 2024).