Of note, the accumulation of Col6A and laminin γ1 mRNAs were increased in the tumors generated by both D2A1-WT and D2A1-KD cells (Supplementary Figures S5E, S5K), suggesting that the presence of tumor cells increased the transcription of these genes in the lung in a non-EMMPRIN-dependent manner. This evidence concerns the gene BSG and neoplasm.