IL15 and steatosis: In the context of MASH, NKT cells not only instigate inflammation and steatosis by releasing LIGHT, IFN-γ, TNF-α, and IL-17A upon recognition of lipid antigens but also motivate HSCs to promote fibrosis via the secretion of OPN and Hh ligands in response to IL-15 stimulation and Hh pathway activation (67, 150, 151).