BECN1 and Alzheimer disease: Consistently, in this research, our data demonstrated that the Beclin-1 and LC3II/LC3I levels were declined while p62 level was elevated in Aβ-treated SH-SY5Y cells, while were partially reversed by LINC00672 overexpression These findings illustrated that overexpression of LINC00672 contributed to the activation of autophagy in AD cell model.