TP53 and metabolic dysfunction-associated steatotic liver disease: These upregulated NAFLD vs. control group genes were implicated in various significant NAFLD-related KEGG pathways, such as protein digestion and absorption, extracellular matrix (ECM)–receptor interaction, amebiasis, p53 signaling pathway, cell cycle, peroxisome proliferator–activated receptor (PPAR) signaling pathway, cytokine-cytokine receptor interaction, and AGE-RAGE signaling pathway in diabetic complications (Figure 1(c)).