KRT17 and neoplasm: Specifically, genes associated with cellular stress, immune responses, and proliferation—including FDCSP, KRT14, and KRT17—were significantly upregulated in MECs from TNBC tissues, while tumor-suppressive genes related to the extracellular matrix (ECM) remodeling and inflammation control—such as CXCL8, SRGN, and DCN—were downregulated [44, 49–54].