STING1 and cervical cancer: STING is a pivotal intracellular receptor that plays a significant role in the immune response.[9] It detects abnormal DNA arising from DNA damage or viral infection, thereby activating TBK1,[10] which subsequently phosphorylates IRF3.[11] This cascade triggers type I interferons (e.g., IFN‐α/β)[12] and initiates antiviral and anti‐tumor responses, including the recruitment of CD8+ T cells through chemokine release.[13] The involvement of the STING pathway in cervical cancer has attracted considerable attention.