In view of these results and the important role that STING plays in innate immunity, it is reasonable to assume that STING‐mediated degradation of HPV E7 is only a part of the effect of the BAG2‐STING axis on the development of cervical cancer, and that BAG2‐STING inhibits the progression of cervical cancer mainly through type I interferon signaling. The gene discussed is STING1; the disease is cervical cancer.