Immunoglobulin A nephropathy (IgAN), described for the first time in 1968 by Berger and Hinglais [1], is considered to be one of the most common forms of glomerulonephritis and one of the main causes of end-stage renal disease (ESRD), which occurs due to damage to the nephrons by the accumulation of immunoglobulin A (IgA) deposits [2,3]. This evidence concerns the gene CD79A and chronic kidney disease.