Although mutations in PLA2G6 (PARK14) were initially correlated with infantile neuroaxonal dystrophy and neurodegeneration with brain iron accumulation, it was subsequently shown that PLA2G6 gene variants correlate with juvenile- and early-onset PD, usually in the early–mid-20s, with atypical features (dystonia–parkinsonism), and characterized by autosomal recessive inheritance [78]. Here, PLA2G6 is linked to Parkinson disease.