Thyroid hormone (TH) attenuates NAFLD by stimulating mitochondrial autophagy and mitochondrial biogenesis [70], with increased mRNA levels of BNIP3, NIX, ULK1, sequestosome 1(p62), and microtubule-associated protein 1A/1B-light chain 3 (LC3) in response to TH. This evidence concerns the gene TH and metabolic dysfunction-associated steatotic liver disease.