Increases in GP73 have been linked to a reduction in type 1 interferon (IFN) and other inflammatory cytokines, whereas soluble GP73 inhibits IL-12 production in monocyte-derived dendritic cells suppressing the antitumor response, whereas overexpression promotes chemokine ligand-2 transcription contributing to tumor metastasis and progression through recruitment of myeloid-derived suppressor cells.4 Here, GOLM1 is linked to neoplasm.