Conversely, SF3B6 expression was significantly negatively correlated with various signaling pathways that influence tumor immunity, including platelet activation, vascular smooth muscle contraction, chemokine signaling, cytokine-cytokine receptor interaction, B cell receptor signaling, viral protein interactions with cytokines and their receptors, malaria, the coagulation complement cascade, and C-type lectin receptor signaling pathways. The gene discussed is SF3B6; the disease is neoplasm.