Studies have shown that H. pylori infection can induce gastric andesophageal inflammation and systemic and vascular inflammation, potentiallyincreasing the risk of AF through increased CRP levels [14].This result is achieved through the activation of the classicalcomplement pathway and the binding of CRP and phosphatidylcholine, the latter ofwhich can cause membrane dysfunction, leading to abnormal sodium and calciumprocessing. Here, CRP is linked to atrial fibrillation.