In line with this, analysis of RNA-seq data collected from skeletal muscles of cancer-free mice with inducible skeletal-muscle-specific FOXP1 overexpression (FoxP1iSkmOE) revealed that FOXP1 overexpression is sufficient to upregulate transcriptional repressors of the clock (Per1, Cry2, and Bhlhe41) and repress clock activators (Bmal1 and Clock) (Figure 5A). The gene discussed is FOXP1; the disease is cancer.