MUC1 and neoplasm: In a glucose‐deficient environment, PKCζ inhibits metabolic reprogramming in colorectal cancer cells and suppresses tumor cell growth.[74] Additionally, PKCζ phosphorylates adenosine deaminase acting on RNA 2 (ADAR2), which inhibits the cellular secretion of miR‐200, thereby suppressing tumor metastasis.[75] Our findings indicate that MUC1 enriches CSLCs by inhibiting PKCζ activity, supporting the tumor suppressor role of PKCζ.