Interestingly, comparing de novo and stringently characterized secondary AMLs emerging after a recorded phase of MDS, the French group discovered a molecular grouping, termed “secondary‐type AML,” defined by mutations in either SRSF2, SF3B1, U2AF1, ZRSR2, ASXL1, EZH2, BCOR, and/or STAG2 genes. Here, SF3B1 is linked to myelodysplastic syndrome.