DCM‐specific interactions, including BMP4‐ACVR1 and BDNF‐NTRK2 (Figure 5C), may collectively reflect the pivotal roles of inflammation—whether resulting from pathogenic infections or autoimmune signalling—in myocarditis, which subsequently leads to DCM [44, 45]. Here, NTRK2 is linked to familial dilated cardiomyopathy.